Years of scientific detective work finally revealed the basis for the non-cell-autonomous effects of brassinosteroid signaling previously suspected by us and others. Our verdict: most likely they don’t exist. Rather, we found that the brassinosteroid receptor gene body contains regulatory elements, which confer ubiquitous expression of trace receptor amounts that are sufficient to promote brassinosteroid-dependent root growth. Our data, therefore, argue for a largely cell-autonomous action of brassinosteroid receptors. Formerly available as a preprint on BioRxiv, now published in Science Advances with additional evidence, controls and textual clarifications.